In free flap surgery, patency of the arterial and venous microvascular anastomoses is a major contributor to flap survival. Extended periods of vascular compromise may lead to ischaemic necrosis, which can only be minimized by tissue reperfusion. However, restoration of blood flow evokes ischaemia reperfusion injury. In this manuscript, a case is presented of a patient who demonstrated a normal oxygenation pattern of a deep inferior epigastric perforator (DIEP) flap. As an ischaemic response can be seen as a sign of temporal vascular compromise, we hypothesize that a short ischaemia time is feasible for tissue viability by eliminating ischaemia reperfusion injury.
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Purpose: The MHC-unrestricted activity of cytokine-induced killer (CIK) cells against chemo-surviving melanoma cancer stem cells (mCSC) was...
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Publication date: 18 April 2017 Source: Cell Reports, Volume 19, Issue 3 Author(s): David Estoppey, Chia Min Lee, Marco Janoschke, Boon He...
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from #AlexandrosSfakianakis via Alexandros G.Sfakianakis on Inoreader http://ift.tt/2oLOayI via IFTTT
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Abstract Functionalised electrospun polyamide-6 (PA-6) nanofibres incorporating gadolinium oxide nanoparticles conjugated to zinc tetracar...
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Cytokine-dependent renewal of stem cells is a fundamental requisite for tissue homeostasis and regeneration. Spermatogonial progenitor cells...
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Abstract This paper addresses the hybrid consensus-based formation keeping problem for nonholonomic mobile robots in the presence of a nov...
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Current treatments for generalized pustular psoriasis are unsatisfactory. We applied recently-developed techniques for transcriptomic analys...
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Downregulation of mouse CCR3 by lentiviral shRNA inhibits proliferation and induces apoptosis of mouse eosinophils. Mol Med Rep. 2016 ...
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