Voltage-gated sodium channels are crucial determinants of neuronal excitability and signaling. Trafficking of the voltage-gated sodium channel NaV1.7 is dysregulated in neuropathic pain. We identify a trafficking program for NaV1.7 driven by hierarchical interactions with posttranslationally modified versions of the binding partner collapsin response mediator protein 2 (CRMP2). The binding...
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Abstract Background Henoch–Schönlein purpura is the most common vasculitis in children. Its long-term prognosis depends on renal involve...
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Abstract Cerebral ischemia leads to multifaceted injury to the brain. A polytherapeutic drug that can be administered immediately after re...
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Publication date: November 2017 Source: Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, Volume 124, Issue 5 from Imaging...
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Abstract Cerebral and systemic organ microvascular pathologies coexist with human Alzheimer’s disease (AD) neuropathology. In this study, w...
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Grandma covered cancer with make up for months Toowoomba Chronicle In Queensland more than 740 people are diagnosed with head and nec...
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A descriptive study of clinical and radiological profile of longitudinal extensive myelitis in a tertiary hospital in Rajasthan, India Publi...
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Lippincott Journals Monthly Update: Otolaryngology Case Studies Comparing Learning Profiles and Response to Instruction in Autism Spectrum...
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Abstract Background To study whether thyroid function was associated with risk of hip or knee replacement due to primary osteoarthritis....
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Abstract Purpose Changes in the appearance of retinal blood vessels are an important indicator for various ophthalmologic and cardiovasc...
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