Involvement of hypoxia-inducing factor-1{alpha}-dependent plasminogen activator inhibitor-1 up-regulation in Cyr61/CCN1-induced gastric cancer cell invasion. [Additions and Corrections]

VOLUME 283 (2008) PAGES 15807–15815This article has been withdrawn by authors Ming-Tsan Lin, I-Hsin Kuo, Cheng-Chi Chang, Chia-Yu Chu, Been-Ren Lin, and Min-Liang Kuo. The same images were used to represent different experimental conditions. In Fig. 1A, lanes 2 and 4 of the HIF-1α DNA gel were duplicated. The HIF-1α DNA gel from Fig. 1A was reused in Fig. 1E in the HIF-1α rCyr61 panel. The GAPDH DNA gel from Fig. 1A was reused in Fig. 1E as GAPDH rCyr61 and IGF-1 panels, Fig. 5A as GAPDH, and Fig. 6B as input, left panel. The HIF-1β immunoblot from Fig. 1A was reused in Fig. 1B as HIF-1β, AGS and TSGH panels, Fig. 1D as HIF-1β, N87 panel, Fig. 1F as HIF-1β, rCyr61 panel, and Fig. 3D as HIF-1β. The tubulin immunoblot from Fig. 1A was reused in Fig. 5B as tubulin, lower panel, and reused in Fig. 5E as tubulin, left panel. In Fig. 1C, lanes 1 and 2 of the HIF-1α immunoblot were reused in lanes 5 and 6. In Fig. 1D, the HIF-1α immunoblot from the N87 panel was reused in Fig. 1F in the HIF-1α IGF-1 panel. In Fig. 1E, lanes 2 and 3 of the HIF-1α DNA gel from the rCyr61 panel were duplicated in lanes 5 and 6 of the same panel. Also in Fig. 1E, the HIF-1α DNA gel from the CoCl2 panel was reused in the IGF-1 panel as HIF-1α. In Fig. 1F, lanes 4 and 5 were duplicated in the HIF-1β immunoblot...

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